In addition, the efo2-1 learn more mutation perturbed hypocotyl elongation, leaf expansion and formation, and stem elongation. EFO1 and EFO2 are both regulated by the circadian clock. Expression and genetic analyses revealed that EFO2 suppresses flowering largely through the action of CONSTANS (CO) and FLOWERING LOCUS T (FT), suggesting that EFO2 is a negative regulator of photoperiodic flowering. The growth defects in efo2-1 were augmented in efo1 efo2, but the induction of FT in the double mutant was comparable to that in efo2-1. Thus, while EFO2 acts as a floral repressor, EFO1 may not be directly involved in flowering,

but the two genes do have overlapping roles in regulating other developmental processes. EFO1 and EFO2 may function collectively to serve as one of the converging points where the signals of growth and flowering intersect.”
“The two-dimensional (2D) spatial electric-field mapping apparatus [Opt. Express 14, 8694 (2006)] plays an important role in

experiments involving metamaterials, such as the verification of free-space and ground-plane invisibility cloaks. However, such an apparatus is valid only for the transverse-electric (TE) mode and is invalid for the transverse-magnetic (TM) mode, as it requires perfectly magnetic conducting (PMC) planes, which A-769662 do not exist in nature. In this paper, we propose a 2D spatial magnetic-field mapping apparatus based on artificial magnetic conductor (AMC) plates. The AMC structure is designed using periodically perfectly electrical conducting

patches with a sub-wavelength size on a dielectric substrate backed with the ground plane, which can simulate a PMC plane. Using two parallel PMC plates to form a TM-wave planar waveguide, we realize the 2D spatial magnetic-field mapping apparatus in order to measure the external and internal magnetic fields of metamaterials. Two types of excitations, a plane-wave source and a magnetic dipole, are used to feed the system. In order to validate the performance of the magnetic-field mapper, two gradient-index metamaterial lenses are measured, and the experimental results are in good agreement with the full-wave Selleckchem Kinase Inhibitor Library simulations. (C) 2011 American Institute of Physics. [doi:10.1063/1.3610519]“
“Hereditary sensory and autonomic neuropathies have different phenotypes. We report 2 cousins with differing clinical courses of a hereditary sensory and autonomic neuropathy. The progressive disease in case 1 is dominated by loss of sensation, autonomic crises, and pain. Case 2 shows loss of sensation, mental retardation, and deafness, clinically similar to patients with hereditary sensory and autonomic neuropathy type II. Detailed molecular studies in case 1 for all known genes that are associated with hereditary sensory and autonomic neuropathies were negative.